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IL-15-mediated downregulation of FOXO1 and upregulation of CXCR6 increases the sensitivity of these cells to metabolic cues such as acetate and extracellular ATP, promoting MHC class I-independent killing of hepatocytes (142)

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Despite the observed reductions in oxidative stress, LipRSV treatment did not restore the BDNF alterations induced by prenatal VPA exposure, reinforcing the notion that redox modulation alone is insufficient to normalize neurobiological pathways associated with ASD